Peptide therapy is tailored to the individual health profile of each patient. Metabolic markers, liver and kidney function, thyroid function, iron stores, and inflammatory status may all be relevant when selecting a programme and planning ongoing medical monitoring. Extended laboratory testing helps the physician identify potential deficiencies and abnormalities before treatment begins, take them into account when developing the protocol, and establish baseline values for subsequent assessment of progress.
The Peptide Check-Up provides the physician with a baseline laboratory profile before therapy begins. The results help identify parameters that require attention, refine the treatment strategy, and monitor the patient’s health throughout the course.
The body functions as an interconnected system, which is why laboratory preparation for peptide therapy covers several areas at once. The check-up assesses blood cell production and inflammatory status, carbohydrate and protein metabolism, liver and kidney function, thyroid profile, and iron metabolism. Additional markers provide information about allergic sensitisation and eosinophilic activity, as well as morning cortisol levels. For patients who smoke, carboxyhaemoglobin is measured to assess the degree of carbon monoxide exposure.
This comprehensive approach allows the physician to interpret the results as a whole and take the patient’s individual health profile into account when developing a personalised protocol.
The programme includes laboratory markers that provide insight into metabolic status and the function of key organs and systems. Interpreting these results together gives the physician a more complete picture of the patient’s health before a peptide protocol is prescribed.
A fundamental assessment of the cellular components of blood. It measures red blood cells, haemoglobin, platelets, and a range of other haematological parameters.
What it shows: patterns of blood cell production, changes in blood cell numbers and characteristics, and laboratory signs of anaemia.
This test identifies the different types of white blood cells present in the blood and their relative proportions. Each type plays a distinct role in the body’s immune defence.
Why it is performed: to help the physician identify changes in the immune response that may occur with infectious, inflammatory, or allergic processes.
Erythrocyte sedimentation rate can change in a range of conditions and is traditionally used as a non-specific marker of inflammatory activity.
What it shows: possible signs of inflammation. The result is interpreted alongside other laboratory findings and clinical information.
CRP is an acute-phase protein that can rise rapidly in response to inflammation.
Why it is performed: to assess current systemic inflammatory activity and complement information provided by other inflammatory markers.
HbA1c reflects the average blood glucose level over approximately the previous 2–3 months. It therefore provides a longer-term view than a single glucose measurement.
What it shows: the state of carbohydrate metabolism over an extended period and potential abnormalities in glycaemic regulation.
This marker reflects average blood glucose over a shorter period, typically the previous 2–3 weeks.
Why it is performed: to complement the assessment of carbohydrate metabolism and provide information about more recent changes in the patient’s glycaemic profile.
Measures the combined concentration of the major proteins in blood plasma. The result is influenced by protein synthesis, intake, distribution, and loss.
What it shows: protein metabolism and provides additional information about nutritional status and protein synthesis.
Albumin accounts for a substantial proportion of plasma proteins. It is produced by the liver and performs numerous functions, including the transport of hormones, fatty acids, and other compounds.
Why it is performed: to assess protein status and provide one of the laboratory indicators of the liver’s protein-synthesising function.
When carbon monoxide enters the body, it binds to haemoglobin and forms carboxyhaemoglobin. Levels are generally higher in people who smoke because of regular exposure to tobacco smoke.
What it shows: the degree of carbon monoxide exposure and its associated impact on oxygen transport.
Creatinine is produced through normal muscle metabolism and is eliminated from the body primarily by the kidneys.
Why it is performed: to assess kidney filtration and calculate the estimated glomerular filtration rate.
Urea is one of the end products of protein metabolism. It is produced in the liver and subsequently excreted by the kidneys.
What it shows: provides additional information about protein metabolism and renal excretory function when considered alongside other biochemical markers.
Uric acid is formed during the breakdown of purines and is eliminated from the body mainly through the kidneys.
Why it is performed: to assess purine metabolism and identify elevated blood uric acid levels.
Cystatin C is produced by cells throughout the body at a relatively constant rate and filtered by the kidneys. This makes it a useful marker for assessing renal filtration.
What it shows: provides additional information about kidney function and can be used to estimate the glomerular filtration rate.
Bilirubin is produced primarily during the natural breakdown of haemoglobin. It is then processed by the liver and excreted into bile.
Why it is performed: to assess bilirubin metabolism and, together with other markers, provide information about liver and biliary system function.
Aspartate aminotransferase is found inside cells in several tissues, including the liver, heart, and skeletal muscles. Its activity in the blood may change when cells are damaged.
What it shows: possible cellular injury. AST is interpreted together with ALT, other biochemical markers, and the clinical picture.
Alanine aminotransferase is present in high concentrations in liver cells and is one of the principal enzymes used in laboratory assessment of hepatocellular health.
Why it is performed: to help identify signs of liver cell injury and determine whether further medical assessment may be appropriate.
This enzyme is present in several tissues. From a diagnostic perspective, its main sources include the liver, biliary system, and bone.
What it shows: when interpreted with other biochemical tests, it helps assess the hepatobiliary system and identify changes that may require further investigation.
GGT is particularly useful as part of a comprehensive laboratory assessment of the liver and biliary tract.
Why it is performed: to help identify biochemical changes that may accompany impaired bile flow and certain forms of liver injury.
Thyroid-stimulating hormone is produced by the pituitary gland and regulates thyroid activity. It is one of the key laboratory markers used to assess thyroid function.
What it shows: regulation of thyroid activity and potential changes in thyroid status.
Free thyroxine is the biologically available fraction of one of the principal hormones produced by the thyroid gland.
Why it is performed: together with TSH, it helps the physician assess thyroid hormone function.
Triiodothyronine is an active thyroid hormone involved in regulating energy metabolism, thermogenesis, and other physiological processes.
What it shows: the concentration of free T3 and provides additional information for the overall assessment of thyroid status.
TIBC reflects the blood’s potential capacity to bind iron. The value is closely related to transferrin, the main protein responsible for transporting iron.
Why it is performed: to assess the iron transport system and help clarify potential abnormalities in iron metabolism.
Ferritin is the body’s principal iron-storage protein. Its concentration is interpreted in the context of inflammatory status because inflammation can increase ferritin independently of iron stores.
What it shows: the body’s iron reserves and can help identify a potential latent iron deficiency.
A plasma protein whose primary role is to bind iron and transport it to tissues.
Why it is performed: to assess the transport component of iron metabolism and complement ferritin and other iron-profile markers.
This marker shows what proportion of available transferrin is already bound to iron. It therefore provides information not only about iron status but also about the amount of iron available for transport to tissues.
What it shows: the degree of transferrin saturation and complements the comprehensive assessment of iron metabolism.
Iron is also important for the function of several enzyme systems. Haem structures form part of cytochrome P450 enzymes, which are involved in the metabolism of many biologically active compounds. For this reason, an extended iron profile is included in laboratory preparation for a personalised peptide protocol.
Total IgE is one of the laboratory markers that can provide additional information about IgE-mediated sensitisation.
Eosinophil cationic protein is released by activated eosinophils, and changes in its concentration may accompany eosinophilic inflammation.
Why they are performed: the results help the physician assess allergic sensitisation and eosinophilic activity alongside symptoms, medical history, and other investigations.
Cortisol secretion follows a circadian rhythm, with concentrations changing throughout the day. The hormone is involved in energy metabolism, regulation of circadian processes, and the body’s physiological response to different forms of stress.
What it shows: the concentration of free cortisol in the morning and provides additional information for assessing hypothalamic-pituitary-adrenal axis function.



